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In the autumn of 2004, on the third anniversary of the Sept. 11 attacks, a retired international corporate lawyer named Gerald Shea drafted that detailed the operations of alleged Israeli spies suspected of surveilling the 9/11 hijackers in the year prior to the attacks. Written in the cold logic of a courtroom complaint, the 166-page memo gathers up most of the evidence available in the public record regarding possible 9/11-related Israeli espionage. Shea, who was educated at Yale and Columbia and worked for 20 years at one of New York’s top law firms, said his purpose in writing the memo was to spur a Congressional investigation “for the sake of U.S. national interest.” “I’m a lawyer first and foremost,” Shea wrote me in an e-mail. “I wanted to separate these difficult factual problems from the politics in the hope that people who have the power and obligation to do so will conduct a public inquiry to resolve the issue.”

Al Felzenberg, spokesman for the 9/11 Public Discourse Project, confirmed that the commission received the memo in mid-September 2004. But Felzenberg noted it was three and a half weeks too late: the commission closed shop on August 21, 2004, its security clearances and investigative power limited by act of Congress. According to Shea, copies of the memo were also sent to the private offices of specific members of the 10-member 9/11 commission. These included: Thomas H. Kean, commission head and now chairman of THK Consulting; commission vice chair Lee H. Hamilton, president and director of the Woodrow Wilson International Center for Scholars; Richard Ben-Veniste, partner in the Washington law firm of Mayer, Brown, Rowe & Maw; Washington, D.C., lawyers Fred Fielding, Jamie S. Gorelick, and Slade Gorton; and Bob Kerrey, former U.S. Senator, now president of the New School for Social Research. (None of the commissioners returned phone calls for comment on the Shea Memo.) Shea also forwarded his report to the offices of members of the Senate Select Committee on Intelligence, including then-committee chair Jay Rockefeller, and Sens. Trent Lott, Olympia Snowe, Chuck Hagel, Richard Durbin, Carl Levin and Evan Bayh. Also included in the mailings, says Shea, were at least six members of the House Permanent Select Committee on Intelligence, including Reps. Peter Hoekstra and Anna Eshoo. For good measure, Shea sent a copy to the office of his Boston senator, Ted Kennedy. He also peppered the news media, submitting copies to the New York Times, the International Herald Tribune, PBS’ Frontline, ABC News’ investigative team at 20/20, and various smaller venues.

With the sole, and notable, exception of the tiny Philadelphia Times-Herald, which ran a brief overview of the memo, the document was ignored. Not a single member of the major media, not a single Commission member or U.S. lawmaker responded, “not even Richard Ben-Veniste, who was in my class at law school,” says Shea, referring to Columbia Law’s 1967 graduating class.

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A Web Archive for the work and writings of Christopher Ketcham (copyright © Christopher Ketcham, all rights reserved)

A new study is showing that recreational use of Viagra is growing rapidly in men under 45 years of age. From 1998 to 2002, the use of Viagra in men under forty-five tripled, says the study, which looked at 5 million insured American males. What this study indicates is that Viagra is being used as a recreational drug, not as a drug to treat a medical condition. Thus, it belongs more in the category of pot, crack, heroin, or meth rather than being a medicinal pharmaceutical. An ideal agent should be rapidly effective, easy to administer, affordable, applicable to a wide range of patients, and minimally toxic. [edit] Pathophysiology buy cialis 20mg Hypertension (high blood pressure) Conclusion The researchers conclude that erectile dysfunction in diabetes is due to a selective defect in the NO mechanisms within the PVN. This defect is a loss in the synthetic enzyme for the production of NO within the neurons of the PVN. Restoring this synthetic enzyme may have a significant therapeutic value for diabetic patients with ED. Might difficulty achieving an erection be related to the normal aging process? Results: Almost one in 10 of the respondents (9.1%) reported erectile dysfunction that lasted for at least one month over the previous year. More than a quarter (27.2%) of respondents were current smokers, with 20.9% smoking ? 20 cigarettes per day, and 6.3% smoking > 20 cigarettes per day. Compared with non-smokers, the adjusted odds ratios for erectile dysfunction were 1.24 (95% confidence interval (CI) 1.01 to 1.52, p ?=? 0.04) for those smoking ? 20 cigarettes per day and 1.39 (95% CI 1.05 to 1.83, p ?=? 0.02) for those smoking > 20 cigarettes per day, after adjusting for other confounding factors. Older age, low level of education, and taking medications for cardiovascular disease were also independently and positively associated with erectile dysfunction. In contrast, moderate alcohol consumption (14 drinks per day) significantly reduced the likelihood of having erectile dysfunction. By the end of the study, the researchers found no significant differences in vision between the men who took the drugs and those who took the placebo. The findings were published in the April issue of the journal Archives of Ophthalmology. Sildenafil should be taken on an empty stomach approximately 30-60 minutes prior to sexual intercourse. This agent is not intended to be taken daily. Sexual stimulation is necessary to produce an erection. An increased ability to obtain erections can last up to 24 hours but is usually limited to 6-8 hours. Call your doctor immediately or go to an emergency room if medication for erection problems give you an unwanted erection that lasts more than 4 hours. Permanent impotence or other lasting damage to your penis may result from this condition. Men with moderately severe liver disease also should not take more than a 5 mg dose of vardenafil in a 24-hour period. Although we have shown that PDE inhibitors reduce NOS2 and ARG1, the full mechanisms underlying these effects remain to be elucidated. One putative mechanism involves the impact of these inhibitors on mRNA stability. cGMP destabilizes NOS2 mRNA by reducing the ubiquitous mRNA binding protein, human antigen R (45). As such, destabilization of NOS2 mRNA via PDE5 inhibition would abrogate NO-mediated immunosuppression more effectively than would competitive inhibition of NO itself. However, because ARG1 mRNA does not possess adenylate/ uridylate-rich elements and has not been described to be stabilized by human antigen R, other mechanisms are likely involved in PDE5-mediated down-regulation of ARG1. One possibility is that high levels of cGMP induced by PDE5 blockade reduce the cytosolic Ca2+ concentration (46), leading to a reduction of the calcium-dependent protein kinase C activity (47) that in turn prevents up-regulation of IL-4R (48). The link between IL-4R and ARG1 in MDSCs is supported by recent data (29) demonstrating a direct correlation between ARG1 expression and IL-4R expression. ACT of tumor-primed CD8 T cells completely eradicated a preestablished C26GM tumor in the LysMCreIL-4R/flox mice in which IL-4R expression has been deleted in neutrophils and macrophages, whereas no effect was seen in the control littermates (29). Our findings support these data by demonstrating that PDE5 blockade down-regulates IL-4R expression on tumor-infiltrating MDSCs (Fig. 6) and synergizes with the adoptive transfer of tumor-primed CD8+ T cells (Fig. 3 and Fig. 4). This effect appears to specifically target MDSCs, because IL-4R expression on isolated CD11b+ cells from tumor-bearing mice is significantly reduced when co-cultured in the presence of sildenafil (Fig. 5). Collectively, these findings underscore the critical role of the IL-4RARG1 pathway in MDSCs, as well as the use of PDE5 inhibitors as therapeutically effective drugs to overcome tumor-induced immunosuppression. Qaseem and colleagues analyzed the findings of 130 studies that evaluated PDE-5 inhibitors alone or combined. They found that treatment with the drugs led to statistically significant and clinically relevant improvements in sexual intercourse and erectile function in men with ED, regardless of the cause (e.g., diabetes, depression, prostate cancer) or ED severity at the start of the study. Besides using enhancement products, there are many other ways to attain virility. The best option is to eliminate bad habits such as smoking and excessive drinking. Better diet and regular exercise also improves sexual life .Today, male enhancement is one of the easiest topics to explore since a lot of study and research are dedicated towards it. Increase in male enhancement research will result in new methods and better remedies. Male Enhancement provides detailed information on Male Enhancement, Male Enhancement Drugs, Male Enhancement Exercise, Male Enhancement Herbs and more. Male Enhancement is affiliated with Cause of Erectile Dysfunctions. Feelings that can lead to erectile dysfunction

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